Treat Magnesium Deficiency by Removing Mercury
Today, clinicians have major concerns with Toxic Body Burden, consisting of toxic heavy metals, pesticides, volatile organic compounds (VOC's) and pathogen load, and how these poisons adversely affect the nutritional status of their patients. A particularly critical example of the interference of toxins with nutritional status and biochemical function is the competitive aspect of Mercury and Magnesium.
Mercury, Magnesium and Adenosine triphosphate (ATP)
Mercury specifically competes with Magnesium and interferes with all Magnesium-dependent metabolic pathways, such as production of energy from ATP and GTP, which directly leads to lack of chemical energy. Every cell in the body requires chemical energy derived from ATP or GTP to function, heal and regenerate. Adenosine-5'-triphosphate (ATP) is a multifunctional nucleotide that is critical as the "molecular currency" of intracellular energy transfer. In this role, ATP transports chemical energy within cells for metabolism. It is produced as an energy source during the processes of photosynthesis and cellular respiration and consumed by many enzymes and a multitude of cellular processes, including biosynthetic reactions, motility and cell division. In signal transduction pathways, ATP is used as a substrate by kinases that phosphorylate proteins and lipids, as well as by adenylate cyclase, which uses ATP to produce the second messenger molecule cyclic AMP. If Mercury is present, this cannot occur.
Signs of Magnesium deficiency include confusion, disorientation, loss of appetite, depression, muscle contractions and cramps, tingling, numbness, abnormal heart rhythms, coronary spasm, migraines and seizures. Numerous illnesses have been associated with Magnesium deficiency including multiple sclerosis, hypertension, insulin resistance, diabetes mellitus, gluten-sensitive enteropathy, premenstrual mood changes, Amyotrophic lateral sclerosis, migraine, rheumatoid arthritis, supraventricular and ventricular arrhythmias, myocardial infarction and sudden coronary death, just to mention a few.
The results of Anner and Moosmayer's research showed that the metal-binding interface of Na-K-ATPase molecule is profoundly implicated in active ion transport and that the intracellular part of the Na-K-ATPase molecule presents the primary target for Mercury action.
Remove the Mercury. Restore Magnesium. Heal the Patient.
The exciting aspect of this body of research for clinicians is that Advanced Cellular Zeolite (ACZ) from Results RNA? is proven to be the superior Mercury detoxifier available as shown in multiple pre- and post- urine provocation studies. Taken orally ACZ is systemically absorbed, removing toxic heavy metals, pesticides, VOC's and free radicals of all types from the body's tissues with highest affinity for Mercury. ACZ does not bind nutrient minerals. This unique action of preferentially binding Mercury and other harmful toxicants without binding nutrient metals allows Magnesium to be absorbed, assimilated and to bind to receptors, so that ATP energetic reactions and other critical Magnesium-dependent pathways can proceed. As an adjunct to Advanced Cellular Zeolite, Advanced Cellular Silver kills pathogens and is itself a nutrient mineral proven to be helpful in cellular healing and regeneration. In my experience, patients can often experience quick, significant improvement in a wide-range of symptomatology through the concomitant use of ACZ and ACS. These exceptional products safely and effectively remove the Toxic Body Burden of toxins and pathogens, which allows the absorption, assimilation and binding of the many nutrients critical for optimal biochemical function and overall health.
ACZ nano and ACS 200 are significantly effective in the systemic removal of toxicants.See the Mercury excretion results of the following pre- and post- urine provocation studies.
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PRE 0.24 
POST 13.05 Beyond removing Mercury, it is important that patients ingest adequate Magnesium through food and supplementation if necessary. According to recent USDA surveys, the average intake of Magnesium by women 19 to 50 years of age is about 74 percent of the Recommended Daily Allowance. Men of the same age had intake of about 94 percent of the recommended daily amount. Approximately 50 percent of women had intakes below 70 percent of the RDA.
Recommended Daily Requirements of Magnesium:
- Children
- 1-3 years old: 80 milligrams
- 4-8 years old: 130 milligrams
- 9-13 years old: 240 milligrams
- 14-18 years old (boys): 410 milligrams
- 14-18 years old (girls): 360 milligrams
- Adult females: 310 milligrams
- Pregnancy: 360-400 milligrams
- Breastfeeding women: 320-360 milligrams
- Adult males: 400 milligram
| Foods High in Magnesium | Serving Size | Magnesium (mg) |
|---|---|---|
| Beans, black | 1 cup | 120 |
| Broccoli, raw | 1 cup | 22 |
| Halibut | 1/2 fillet | 170 |
| Nuts, peanuts | 1 oz | 64 |
| Okra, frozen | 1 cup | 94 |
| Oysters | 3 oz | 49 |
| Plantain, raw | 1 medium | 66 |
| Rockfish | 1 fillet | 51 |
| Scallop | 6 large | 55 |
| Seeds, pumpkin and squash | 1 oz (142 seeds) | 151 |
| Soy milk | 1 cup | 47 |
| Spinach, cooked | 1 cup | 157 |
| Tofu | 1/4 block | 37 |
| Whole grain cereal, ready-to-eat | 3/4 cup | 24 |
| Whole grain cereal, cooked | 1 cup | 56 |
| Whole wheat bread | 1 slice | 24 |
Source: USDA Nutrient Database for Standard References, Release 15 for Magnesium
References
1-Thompson JD, Nechay BR. Inhibition by metals of canine renal calcium, magnesium-activated adenosinetriphosphatase. J Toxicol Environ Health. 1981 Jun;7(6):901-8. PubMed PMID: 6115068.
2-Araujo GM, Silva CB, Hasson-Voloch A. Comparison of the inhibitory effects of mercury and cadmium on the creatine kinase from Electrophorus electricus (L). Int J Biochem Cell Biol. 1996 Apr;28(4):491-7. PubMed PMID: 9026360.
3-Chetty CS, McBride V, Sands S, Rajanna B. Effects in vitro of mercury on rat brain Mg(++)-ATPase. Arch Int Physiol Biochim. 1990 Oct;98(5):261-7. PubMed PMID: 1708994.
4-Milosevi? M, Petrovi? S, Demajo M, Horvat A. Effects of metal ions on plasma membrane Mg2+-atpase in rat uterus and ovaries. Ann N Y Acad Sci. 2005 Jun;1048:445-8. PubMed PMID: 16154973.
5-Anner, BM, Moosmayer,M. Mercury inhibits Na-K-ATPase primarily at the cytoplasmic side. AmJPhysiology. Vol 262, Issue 5 p843-848, 1992
6-Moreira,CM, Oliveira,EM, Bonan,CD, Sarkis,JJF and Vassallo, DV. Effects of Mercury on myosin ATPase in the ventricular myocardium of the rat. Comparative biochemistry and physiology. Toxicology & pharmacology : CBP 2003;135C(3):269-75
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21-Journal of the American Academy of Nurse Practitioners. December 2009, Volume 21, Issue 12, Pages: 651-657 "Oral magnesium supplementation in adults with coronary heart disease or coronary heart disease risk"